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Circulating Levels of Tumor Necrosis Factor-Alpha Receptor 2 Are Increased in Heart Failure with Preserved Ejection Fraction Relative to Heart Failure with Reduced Ejection Fraction: Evidence for a…

Overview of attention for article published in PLOS ONE, June 2014
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Title
Circulating Levels of Tumor Necrosis Factor-Alpha Receptor 2 Are Increased in Heart Failure with Preserved Ejection Fraction Relative to Heart Failure with Reduced Ejection Fraction: Evidence for a Divergence in Pathophysiology
Published in
PLOS ONE, June 2014
DOI 10.1371/journal.pone.0099495
Pubmed ID
Authors

Brendan N. Putko, Zuocheng Wang, Jennifer Lo, Todd Anderson, Harald Becher, Jason R. B. Dyck, Zamaneh Kassiri, Gavin Y. Oudit

Abstract

Various pathways have been implicated in the pathogenesis of heart failure (HF) with preserved ejection fraction (HFPEF). Inflammation in response to comorbid conditions, such as hypertension and diabetes, may play a proportionally larger role in HFPEF as compared to HF with reduced ejection fraction (HFREF).

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The data shown below were collected from the profile of 1 X user who shared this research output. Click here to find out more about how the information was compiled.
Mendeley readers

Mendeley readers

The data shown below were compiled from readership statistics for 108 Mendeley readers of this research output. Click here to see the associated Mendeley record.

Geographical breakdown

Country Count As %
China 1 <1%
Brazil 1 <1%
Unknown 106 98%

Demographic breakdown

Readers by professional status Count As %
Student > Ph. D. Student 23 21%
Student > Bachelor 14 13%
Researcher 12 11%
Other 10 9%
Student > Postgraduate 9 8%
Other 18 17%
Unknown 22 20%
Readers by discipline Count As %
Medicine and Dentistry 45 42%
Biochemistry, Genetics and Molecular Biology 15 14%
Agricultural and Biological Sciences 7 6%
Immunology and Microbiology 2 2%
Sports and Recreations 2 2%
Other 9 8%
Unknown 28 26%