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TRAIL Deficiency Contributes to Diabetic Nephropathy in Fat-Fed ApoE-/- Mice

Overview of attention for article published in PLOS ONE, March 2014
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Title
TRAIL Deficiency Contributes to Diabetic Nephropathy in Fat-Fed ApoE-/- Mice
Published in
PLOS ONE, March 2014
DOI 10.1371/journal.pone.0092952
Pubmed ID
Authors

Siân P. Cartland, Jonathan H. Erlich, Mary M. Kavurma

Abstract

We recently demonstrated that TNF-related apoptosis-inducing ligand (TRAIL) is protective of diet-induced diabetes in mice. While TRAIL has been implicated in chronic kidney disease, its role in vivo in diabetic nephropathy is not clear. The present study investigated the role of TRAIL in the pathogenesis of diabetic nephropathy using TRAIL(-/-)ApoE(-/-) mice.

Mendeley readers

Mendeley readers

The data shown below were compiled from readership statistics for 26 Mendeley readers of this research output. Click here to see the associated Mendeley record.

Geographical breakdown

Country Count As %
Unknown 26 100%

Demographic breakdown

Readers by professional status Count As %
Researcher 5 19%
Student > Doctoral Student 4 15%
Student > Bachelor 3 12%
Student > Postgraduate 3 12%
Student > Ph. D. Student 2 8%
Other 6 23%
Unknown 3 12%
Readers by discipline Count As %
Medicine and Dentistry 7 27%
Agricultural and Biological Sciences 5 19%
Biochemistry, Genetics and Molecular Biology 3 12%
Immunology and Microbiology 2 8%
Social Sciences 1 4%
Other 1 4%
Unknown 7 27%