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Human A53T α-Synuclein Causes Reversible Deficits in Mitochondrial Function and Dynamics in Primary Mouse Cortical Neurons

Overview of attention for article published in PLOS ONE, December 2013
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1 CiteULike
Title
Human A53T α-Synuclein Causes Reversible Deficits in Mitochondrial Function and Dynamics in Primary Mouse Cortical Neurons
Published in
PLOS ONE, December 2013
DOI 10.1371/journal.pone.0085815
Pubmed ID
Authors

Li Li, Sashi Nadanaciva, Zdenek Berger, Wei Shen, Katrina Paumier, Joel Schwartz, Kewa Mou, Paula Loos, Anthony J. Milici, John Dunlop, Warren D. Hirst

Mendeley readers

Mendeley readers

The data shown below were compiled from readership statistics for 112 Mendeley readers of this research output. Click here to see the associated Mendeley record.

Geographical breakdown

Country Count As %
Luxembourg 1 <1%
Unknown 111 99%

Demographic breakdown

Readers by professional status Count As %
Student > Ph. D. Student 21 19%
Researcher 20 18%
Student > Bachelor 14 13%
Student > Master 13 12%
Student > Doctoral Student 6 5%
Other 15 13%
Unknown 23 21%
Readers by discipline Count As %
Biochemistry, Genetics and Molecular Biology 26 23%
Neuroscience 23 21%
Agricultural and Biological Sciences 22 20%
Medicine and Dentistry 11 10%
Pharmacology, Toxicology and Pharmaceutical Science 2 2%
Other 4 4%
Unknown 24 21%