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The Multifunctional Ca2+/Calmodulin-Dependent Kinase IIδ (CaMKIIδ) Regulates Arteriogenesis in a Mouse Model of Flow-Mediated Remodeling

Overview of attention for article published in PLOS ONE, August 2013
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Title
The Multifunctional Ca2+/Calmodulin-Dependent Kinase IIδ (CaMKIIδ) Regulates Arteriogenesis in a Mouse Model of Flow-Mediated Remodeling
Published in
PLOS ONE, August 2013
DOI 10.1371/journal.pone.0071550
Pubmed ID
Authors

Jason A. Scott, Paula J. Klutho, Ramzi El Accaoui, Emily Nguyen, Ashlee N. Venema, Litao Xie, Shuxia Jiang, Megan Dibbern, Sabrina Scroggins, Anand M. Prasad, Elisabeth D. Luczak, Melissa K. Davis, Weiwei Li, Xiaoqun Guan, Johannes Backs, Annette J. Schlueter, Robert M. Weiss, Francis J. Miller, Mark E. Anderson, Isabella M. Grumbach

Abstract

Sustained hemodynamic stress mediated by high blood flow promotes arteriogenesis, the outward remodeling of existing arteries. Here, we examined whether Ca²⁺/calmodulin-dependent kinase II (CaMKII) regulates arteriogenesis.

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Mendeley readers

The data shown below were compiled from readership statistics for 22 Mendeley readers of this research output. Click here to see the associated Mendeley record.

Geographical breakdown

Country Count As %
Unknown 22 100%

Demographic breakdown

Readers by professional status Count As %
Researcher 4 18%
Student > Bachelor 3 14%
Student > Master 3 14%
Other 2 9%
Professor 2 9%
Other 3 14%
Unknown 5 23%
Readers by discipline Count As %
Medicine and Dentistry 6 27%
Agricultural and Biological Sciences 4 18%
Biochemistry, Genetics and Molecular Biology 3 14%
Pharmacology, Toxicology and Pharmaceutical Science 1 5%
Neuroscience 1 5%
Other 1 5%
Unknown 6 27%