Florence Burté, Biobele J. Brown, Adebola E. Orimadegun, Wasiu A. Ajetunmobi, Francesca Battaglia, Barry K. Ely, Nathaniel K. Afolabi, Dimitrios Athanasakis, Francis Akinkunmi, Olayinka Kowobari, Samuel Omokhodion, Kikelomo Osinusi, Felix O. Akinbami, Wuraola A. Shokunbi, Olugbemiro Sodeinde, Delmiro Fernandez-Reyes

Cerebral malaria (CM) and severe malarial anemia (SMA) are the most serious life-threatening clinical syndromes of Plasmodium falciparum infection in childhood. Therefore it is important to understand the pathology underlying the development of CM and SMA, as opposed to uncomplicated malaria (UM). Different host responses to infection are likely to be reflected in plasma proteome-patterns that associate with clinical status and therefore provide indicators of the pathogenesis of these syndromes.