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The Transcriptional Targets of Mutant FOXL2 in Granulosa Cell Tumours

Overview of attention for article published in PLOS ONE, September 2012
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Title
The Transcriptional Targets of Mutant FOXL2 in Granulosa Cell Tumours
Published in
PLOS ONE, September 2012
DOI 10.1371/journal.pone.0046270
Pubmed ID
Authors

Roseanne Rosario, Hiromitsu Araki, Cristin G. Print, Andrew N. Shelling

Abstract

Despite their distinct biology, granulosa cell tumours (GCTs) are treated the same as other ovarian tumours. Intriguingly, a recurring somatic mutation in the transcription factor Forkhead Box L2 (FOXL2) 402C>G has been found in nearly all GCTs examined. This investigation aims to identify the pathogenicity of mutant FOXL2 by studying its altered transcriptional targets.

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The data shown below were compiled from readership statistics for 41 Mendeley readers of this research output. Click here to see the associated Mendeley record.

Geographical breakdown

Country Count As %
Unknown 41 100%

Demographic breakdown

Readers by professional status Count As %
Student > Ph. D. Student 12 29%
Researcher 9 22%
Student > Master 4 10%
Professor > Associate Professor 3 7%
Student > Bachelor 3 7%
Other 5 12%
Unknown 5 12%
Readers by discipline Count As %
Agricultural and Biological Sciences 13 32%
Biochemistry, Genetics and Molecular Biology 10 24%
Medicine and Dentistry 9 22%
Psychology 1 2%
Unspecified 1 2%
Other 0 0%
Unknown 7 17%