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FAK Regulates Intestinal Epithelial Cell Survival and Proliferation during Mucosal Wound Healing

Overview of attention for article published in PLOS ONE, August 2011
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Title
FAK Regulates Intestinal Epithelial Cell Survival and Proliferation during Mucosal Wound Healing
Published in
PLOS ONE, August 2011
DOI 10.1371/journal.pone.0023123
Pubmed ID
Authors

Katherine A. Owen, Michelle Y. Abshire, Robert W. Tilghman, James E. Casanova, Amy H. Bouton

Abstract

Following damage to the intestinal epithelium, restoration of epithelial barrier integrity is triggered by a robust proliferative response. In other tissues, focal adhesion kinase (FAK) regulates many of the cellular processes that are critical for epithelial homeostasis and restitution, including cell migration, proliferation and survival. However, few studies to date have determined how FAK contributes to mucosal wound healing in vivo.

Mendeley readers

Mendeley readers

The data shown below were compiled from readership statistics for 80 Mendeley readers of this research output. Click here to see the associated Mendeley record.

Geographical breakdown

Country Count As %
United States 3 4%
Unknown 77 96%

Demographic breakdown

Readers by professional status Count As %
Student > Ph. D. Student 25 31%
Researcher 12 15%
Student > Master 8 10%
Student > Bachelor 6 8%
Professor > Associate Professor 4 5%
Other 10 13%
Unknown 15 19%
Readers by discipline Count As %
Agricultural and Biological Sciences 32 40%
Biochemistry, Genetics and Molecular Biology 12 15%
Medicine and Dentistry 11 14%
Pharmacology, Toxicology and Pharmaceutical Science 2 3%
Nursing and Health Professions 1 1%
Other 6 8%
Unknown 16 20%