| Title |
PINK1 Defect Causes Mitochondrial Dysfunction, Proteasomal Deficit and α-Synuclein Aggregation in Cell Culture Models of Parkinson's Disease
|
|---|---|
| Published in |
PLOS ONE, February 2009
|
| DOI | 10.1371/journal.pone.0004597 |
| Pubmed ID | |
| Authors |
Wencheng Liu, Cristofol Vives-Bauza, Rebeca Acín-Peréz-, Ai Yamamoto, Yingcai Tan, Yanping Li, Jordi Magrané, Mihaela A. Stavarache, Sebastian Shaffer, Simon Chang, Michael G. Kaplitt, Xin-Yun Huang, M. Flint Beal, Giovanni Manfredi, Chenjian Li |
Mendeley readers
The data shown below were compiled from readership statistics for 133 Mendeley readers of this research output. Click here to see the associated Mendeley record.
Geographical breakdown
| Country | Count | As % |
|---|---|---|
| United Kingdom | 1 | <1% |
| United States | 1 | <1% |
| Czechia | 1 | <1% |
| Germany | 1 | <1% |
| Unknown | 129 | 97% |
Demographic breakdown
| Readers by professional status | Count | As % |
|---|---|---|
| Student > Ph. D. Student | 27 | 20% |
| Researcher | 21 | 16% |
| Student > Master | 18 | 14% |
| Student > Bachelor | 13 | 10% |
| Other | 7 | 5% |
| Other | 25 | 19% |
| Unknown | 22 | 17% |
| Readers by discipline | Count | As % |
|---|---|---|
| Agricultural and Biological Sciences | 46 | 35% |
| Neuroscience | 21 | 16% |
| Biochemistry, Genetics and Molecular Biology | 19 | 14% |
| Medicine and Dentistry | 7 | 5% |
| Pharmacology, Toxicology and Pharmaceutical Science | 5 | 4% |
| Other | 6 | 5% |
| Unknown | 29 | 22% |